Gene content    
KCNK9 ( by HUGO)
Potassium Channel, Subfamily K, Member 9
Oncogene
Potassium Channel
Subfamily K
Member 9
TASK3
Acid-Sensitive Potassium Channel Protein TASK-3
Two Pore K(+) Channel KT3.2
Two Pore Potassium Channel KT3.2
TWIK-Related Acid-Sensitive K(+) Channel 3
K2p9.1
KT3.2
TASK-3
Potassium Channel Subfamily K Member 9
NCBI: 8q24.3    Ensembl: 8q24.3
KCNK9_HUMANSize: 374 amino acidsMass: 42264 Da

  • Tissue specificity: Mainly found in the cerebellum. Also found in adrenal gland, kidney and lung [IMAGE] Custom PCR Arrays for KCNK9 Primer Products: [IMAGE] OriGene qSTAR qPCR primer pairs in human, mouse for KCNK9 [IMAGE] Pre-validated RT2 qPCR Primer Assay in human, mouse
  • Function:
    UniProtKB/Swiss-Prot Summary: KCNK9_HUMAN, Q9NPC2 Function: pH-dependent, voltage-insensitive, background potassium channel protein Gene Ontology (GO): 4 molecular function terms: About this table GO ID Qualified GO term Evidence PubMed IDs GO:0005244 voltage-gated ion channel activity IEA -- GO:0005267 potassium channel activity IEA -- GO:0042803 protein homodimerization activity IEA -- GO:0046982 protein heterodimerization activity IEA -- [IMAGE] Find genes that share ontologies with KCNK9 About GenesLikeMe Phenotypes: 4 GenomeRNAi human phenotypes for KCNK9: Decreased Tat-dependent transc Decreased p24 protein expressi Increased gamma-H2AX phosphory Increased resistance to MDM2 i
  • Similarity:
    Belongs to the two pore domain potassium channel (TC 1.A.1.8) family [IMAGE]

    • Protein Domain/Family    
    Source ID Domain Name Type
    InterProIPR0030922pore_dom_K_chnl_TASKPotassium channel TASKFamily
    IPR0032802pore_dom_K_chnlK+ channel, two poreFamily
    IPR005407KCNK9TASK-3 K+ channelFamily
    BlocksIPB003092TASK K+ channel signature IPB003280 Two pore domain K+ channel signature IPB005407 TASK-3 K+ channel signature IPB013099 Ion transport 2TASK K+ channel signature
    IPB003092TASK K+ channel signatureTASK K+ channel signature
    IPB005407TASK-3 K+ channel signatureTASK-3 K+ channel signature

    Gene Ontology    
    Type Term Evidence Source Pub
    Biological Process potassium ion transport NAS GOA 11042359

    Disorder & Mutation    
    Source Disease
    SWISS-PROTBirk-Barel mental retardation dysmorphism syndrome (BIBAS) [MIM:612292]: A syndrome characterized by mental retardation, hypotonia, hyperactivity, and facial dysmorphism. Note=The disease is caused by mutations affecting the gene represented in this entry

    KCNK9 cross reference    
    PubMed OMIM Entrez Gene NCKU SNP Nucleotide UniProt Genome Data Viewer HomoloGene